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the opportunity to rescue animals from sepsis when cure is specified therapeutically instead of prophylactically is significant, as anti-sepsis therapy, by definition, is always administered following the onset of an infection in human beings. Recapitulating some of these conclusions, Mif-deficient mice were documented to be resistant to endotoxic
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MIF-2 in distinction to MIF appears to absence the mandatory CXCR-interacting motifs needed for activation, and it's considered to exert a far more selective motion in activating the tissue-protecting CD74 signaling pathway. That said, MIF triggers the CD74/CD44/AMPK receptor signaling pathway, which encourages glucose uptake in cardiomyocytes and